A CD4 + T cell-fibroblast crosstalk exacerbates autoimmunity in a mouse model of primary Sjögren disease

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A CD4 + T cell-fibroblast crosstalk exacerbates autoimmunity in a mouse model of primary Sjögren disease

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Small-molecule inhibitors for β-arrestin proteins

Immune cell–fibroblast crosstalk may may drive progression of autoimmune diseases

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