Publications

USP16 S-nitrosylation aggravates coronary microembolization-induced myocardial injury via repressing KDM1A-mediated glutathione homeostasis

 42
USP16 S-nitrosylation aggravates coronary microembolization-induced myocardial injury via repressing KDM1A-mediated glutathione homeostasis

Tags:

Previous Article

Tapt1 gene is essential maintainer of protein synthesis and brain development

Next Article

Driver of cardiac injury after coronary microembolization

Related Posts

Advances in cell therapy: progress and challenges in hematological and solid tumors

Advances in cell therapy: progress and challenges in he...

 112

Higher-order connectomics of human brain function reveals local topological signatures of task decoding, individual identification, and behavior

Higher-order connectomics of human brain function revea...

 77

Androgen receptor signalling in non-prostatic malignancies: challenges and opportunities

Androgen receptor signalling in non-prostatic malignanc...

 90