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USP16 S-nitrosylation aggravates coronary microembolization-induced myocardial injury via repressing KDM1A-mediated glutathione homeostasis

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USP16 S-nitrosylation aggravates coronary microembolization-induced myocardial injury via repressing KDM1A-mediated glutathione homeostasis

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Tapt1 gene is essential maintainer of protein synthesis and brain development

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Driver of cardiac injury after coronary microembolization

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