Multiomic analyses direct hypotheses for Creutzfeldt- Jakob disease risk genes

98

Multiomic analyses direct hypotheses for Creutzfeldt- Jakob disease risk genes

Tags:

Previous Article

Mitochondrial morphology and cellular physiology

Analysis of CreutzfeldtJakob disease risk genes

Related Posts

TRPM2 overactivation drives hyperlipidemia- induced dysfunction of myeloid cells and neurovascular units

TRPM2 overactivation drives hyperlipidemia- induced dys...

149

Gene therapy for epilepsy targeting neuropeptide Y and its Y2 receptor to dentate gyrus granule cells

Gene therapy for epilepsy targeting neuropeptide Y and ...

118

Repeated fasting events sensitize enhancers, transcription factor activity and gene expression to support augmented ketogenesis

Repeated fasting events sensitize enhancers, transcript...

101