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Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypes

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Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypes

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A nucleosome switch primes hepatitis B virus infection

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Reversing fragile X syndrome symptoms by modifying NMDA receptor signaling

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